Blocking Spike in Stem Cell Growth After a Brain Injury

There are about 2 million Americans who suffer from Traumatic Brain Injuries (TBI) annually. The severity of the trauma may differ from patient to patient but leads to a wide range of motor, behavioral, cognitive as well as intellectual disabilities in the patients. These disabilities may be long term or short term. TBI is a global health concern and despite the huge number of injuries, the treatment options are limited. There have been many treatments and studies that suggested excessive burst of new brain cells after TBI helped in the recovery of the patients better. Breaking this common assumption, Dr. Viji Santhakurmar (Associate Professor in the Department of Pharmacology, Physiology and Neuroscience, Rutgers University) and her colleagues proved otherwise with their report. They challenged this assumption and found that an excessive neurogenesis may, in fact, lead to memory decline, seizures. Neurogenesis aftera a Brain Injury May Lead to Memory Decline, Seizures It has been proposed that increase in neurogenesis aids in the repair of the injured brain network. However, survivors of a traumatic brain injury often develop life-changing disorders such as a decrease in memory as well as epileptic seizures. Researchers found there was a considerate increase in the new nerve cell growth (neurogenesis) that was probably to help replace the damaged or destroyed brain cells. However, Santhakumar and her colleagues found evidence contradictory to the popular belief. They found the spike in brain cell growth may, in fact, lead to post-injury seizures and long-term memory decline. This team from Reuters University examined brain injuries in lab rats and found that brain cells at the injury site play an important role. These cells double in number within 3 days after injury; however, they decrease by more than half after a month as compared to rats with no brain injury. The neural stem cells develop into mature cells too showed a similar increase and decrease pattern. Thus, proving they were responsible for the loss of brain cells. wy2hFbmm
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